Research from University of California, Davis, highlights dangers of nanoplastics on glucose metabolism and liver health in mammals.
Ingestion of polystyrene nanoplastics disrupts glucose regulation and shows signs of liver injury in animal studies.
Nanoplastics from plastic degradation enter food chains, posing health risks with estimated human ingestion of thousands to millions of particles annually.
Experimental study reveals systemic glucose intolerance and elevated liver enzyme activity in mice exposed to nanoplastics.
Increased gut permeability in exposed mice leads to bacterial endotoxin translocation, impacting liver health and inflammation.
Findings suggest nanoplastics affect metabolic and hepatic functions, emphasizing the need for further research and regulatory attention.
Collaboration with experts in mass spectrometry aims to investigate nanoplastic accumulation and metabolic disruptions at tissue level.
Caution advised on generalizing preliminary findings before thorough validation, as study was presented at a conference and not yet peer-reviewed.
Understanding biological impacts of micro- and nanoplastics is crucial for regulatory agencies and public health in setting safety thresholds.
Association between nanoplastics and metabolic disorders signals potential risks for diabetes and liver disease in human populations.